And what does insulin have to do with cardiovascular disease.
Insulin resistance is not a strange word out of the dictionary anymore. It has unfortunately become part of our everyday lingo. Just like diabesity.
When you eat a diet rich in sweets and carbs here is what happens;
Your body has to constantly produce large quantities of insulin to make sure your blood sugar does not rise too high, and sends you in a coma, so the insulin is called in to put the sugar away in the cells…(by turning it into fat for storage).
When an hormone like insulin constantly flood your cells, is like constantly screaming in someone’s ear…eventually they will get a bit deaf…so your cells become deaf to insulin and your body needs to produce more and more to keep putting that sugar away….
That eventually leads to insulin resistance.
What does this have to do with cardiovascular disease?
So where do you think all that sugar goes when it gets shuttled away from it’s insulin “cop”?
It really has only two options: One it gets burned for energy in the cells, like when you run a marathon or work out, or play a sport.
Option two is the sugar gets converted to tryglicerides to get stored…for later use. Later use, meaning the time when you will have NOT ENOUGH sugar in your bloodstream and NO insulin to store it away, and fat will be able to be used for energy.
But really, when does that happen? If you keep eating carbs and sugar (and calories), there is always a fresh source of sugar to put in circulation in your blood stream. And if you run out for a moment, before you can ever get to those fat resources, the drop in blood sugar will make you run for more carbs in a hurry!
Tryglicerides, Cholesterol and Inflammation
So why do we have high tryglicerides? Because of the constant influx of carbs!
Why do we have high cholesterol then? Because the body is in a state of chronic inflammation. High insulin, starts a domino effect in the body that leads to a chronic inflammation.
That also means chronically inflamed arteries. In fact the only place where you find cholesterol plaquing is in high flow, high pressure areas like arteries.
So you these inflamed, high flow, high pressure areas that are your arteries, can easily rupture and start leaking….
This is where your body’s natural wisdom, and self repair mechanisms spring into action. The body then sends a repair material to fix the weak area of the artery; This is a strong, flexible, waxy material = cholesterol plaques!
Imagine there is a fire just breaking out in a house, so the neighbor calls the fire department who sends out a fire truck with 10 firemen ready to tackle the problem.
Except when they get there and get to work a strange back car shows up and a bunch of shady guys in black get out and start shooting on them…once they kill them all they go and break down the fire trucks, then leave!
So Cholerstrol is the firemen and LDLs and HDLs are the fire trucks carrying them to the site of the damage.
Can you guess what are the guys in black??
What happens next?
So do you know wonder what happens to the arteries after we take away their ability to patch up damacge with cholesterol? They have no choice but to start calcifying.
Sure, the plaque will become a problem if it keeps building…but now the million dollar question: Why not remove the underlying cause of inflammation and just prevent the problem from starting in the first place? Or if the problem started why not just revert it by eliminating the inflammation?
I would be delighted to hear your thought on this one…please leave a comment below and let’s talk about it!